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Clinical Approach to the Diagnosis of AKI

A structured, evidence-based walkthrough of AKI diagnosis — from KDIGO definition through urinary indices, microscopy, imaging, biomarkers, biopsy, and common diagnostic pitfalls.

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Clinical Approach to the Diagnosis of AKI: Overview

A structured framework for evaluating suspected AKI — definition, KDIGO criteria, and the initial differential.

Definition (KDIGO)

  • AKI = abrupt (hours–days) reduction in kidney function with retention of nitrogenous waste and dysregulation of fluid/electrolyte/acid-base.
  • Diagnostic criteria: ↑ SCr ≥0.3 mg/dL within 48 h, or ≥1.5× baseline within 7 days, or urine output <0.5 mL/kg/h for ≥6 h.
  • Baseline creatinine is critical — estimate from the most recent stable value, not the admission value.

Initial Structured Approach

  • Confirm AKI (vs chronic or AKI-on-CKD) — compare to baseline and trend the creatinine.
  • Stage severity (KDIGO 1–3) — guides monitoring intensity and dialysis threshold.
  • Classify etiology into Pre-renal, Intrinsic (intra-renal), Post-renal — the classic triage.
  • Identify reversible contributors early: hypovolemia, nephrotoxins, obstruction, sepsis.

Red Flags Prompting Urgent Action

  • Hyperkalemia (K⁺ ≥6.0 or ECG changes), severe acidosis (pH <7.1), volume overload with hypoxemia.
  • Uremic symptoms (pericarditis, encephalopathy), anuria, or rapidly rising creatinine.
  • Suspected obstruction with infection, or rapidly progressive glomerulonephritis.
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